Angina pectoris is a syndrome that is characterized by breakthrough pain in the chest due to transient myocardial ischemia. However ischemic episode is short and clinical manifestations of heart attack Absent.
Typical of stable angina is receiving ischemic attack after making a different type of physical activity or mental stress. In this case, rapid heartbeat and elevated blood pressure, increased myocardial oxygen requirements. The pain lasts a few minutes and goes after cessation of exercise or receiving nitroglycerin and other nitrates.
In contrast to stable angina unstable at typical several clinical scenarios. Characterized by increasing the frequency ostratata, strength and duration of ischemic attacks. It is considered that in the pathogenesis of ischemic episodes include other mechanisms – vasoconstriction and formation of platelet aggregation.
In most cases, the attacks are not preceded by an increase in blood pressure and fast heart rate. In certain patients there is a critical narrowing of the vascular lumen due to severe progressive atherosclerosis. In the majority of patients no clear correlation between the degree of stenotic and severity of the clinical course.
Atypical angina Prinzmetal’s is characterized by frequent episodes of pain. They occur most often on holiday at the same time of day. In coronary angiography revealed no atherosclerotic changes. A subset of patients, however, there are serious changes affecting some vessels. The various forms of angina pectoris resulting from various combinations of atherosclerotic coronary stenosis and vasospasm.